Explore our database of over 4000 laboratory markers.
Search and Understand 4000+ Biomarkers
Optimal range: 0 - 29 mmol/mol creatinine
Breakdown product of hyaluronic acid; also found in certain foods.
Optimal range: 0 - 30 mmol/mol
Breakdown product of hyaluronic acid; also found in certain foods.
Optimal range: 0 - 56 mmol/mol creatinine
Produced by action of Candida hyaluronidase on the intercellular cement, hyaluronic acid. Oxidation of the hyaluronic acid breakdown by white blood cell hypochlorite produces tartaric acid and arabinose. Antifungal treatment and high-potency, multi-strain probiotics may help rebalance GI flora.
Optimal range: 0 - 20 mmol/mol creatinine
Breakdown product of hyaluronic acid; also found in certain foods.
Optimal range: 0 - 50 mmol/mol creatinine
Produced by action of Candida hyaluronidase on the intercellular cement, hyaluronic acid. Oxidation of the hyaluronic acid breakdown by white blood cell hypochlorite produces tartaric acid and arabinose. Antifungal treatment and high-potency, multi-strain probiotics may help rebalance GI flora.
Optimal range: 1.3 - 4.7 µmol/L
Arachidic acid (also called eicosanoic acid) is a long-chain saturated fatty acid. It is the elongation product of stearic acid and can be utilized as an energy source to build membranes.
Optimal range: 0.22 - 0.35 wt %
Arachidic acid (also called eicosanoic acid) is a long-chain saturated fatty acid. It is the elongation product of stearic acid and can be utilized as an energy source to build membranes.
Optimal range: 0.1 - 0.53 %
Arachidic acid (also called eicosanoic acid) is a long-chain saturated fatty acid. It is the elongation product of stearic acid and can be utilized as an energy source to build membranes.
Optimal range: 0.24 - 0.4 wt %
Arachidic acid is very long, 20-carbon backbone saturated fatty acid (20:0). It is found in various nuts, soybeans, peanut oil, corn oil, and cocoa butter. In addition to dietary sources, it can be synthesized by the hydrogenation of the omega-6 fatty acid arachidonic acid or the elongation of stearic acid.
Optimal range: 158 - 521 µmol/L
Arachidonic acid is an inflammatory omega-6 fatty acid. Our bodies produce this nutrient, and its excess may lead to inflammatory diseases and mood disorders.
Optimal range: 15 - 21 wt %
Arachidonic acid is an inflammatory omega-6 fatty acid. Our bodies produce this nutrient, and its excess may lead to inflammatory diseases and mood disorders.
Optimal range: 7 - 12 wt %
Arachidonic acid (AA) is a 20-carbon polyunsaturated n-6 fatty acid with 4 double bonds (20:4n6). Its double bonds contribute to cell membrane fluidity and predispose it to oxygenation. This can lead to several important metabolites which ensure a properly functioning immune system as well as regulate inflammation, brain activity, and other signaling cascades. AA’s metabolites are called eicosanoids which are signaling molecules. They can be produced via cyclooxygenases, lipoxygenase, cytochrome P450, and oxygen species-triggered reactions. These pathways yield molecules like prostaglandins, isoprostanes, thromboxane, leukotrienes, lipoxins, and epoxyeicosatrienoic acids. AA can be obtained in the diet from eggs, fish, and animal meats and fats – or produced directly from DGLA using the delta-5-desaturase enzyme. Although often vilified, adequate AA intake is needed to achieve an equilibrium between its inflammatory and resolution effects to support a healthy immune system. It is also fortified in infant formulas due to its importance in growth and development.
Optimal range: 8.6 - 15.6 % by wt
Arachidonic acid is an inflammatory omega-6 fatty acid. Our bodies produce this nutrient, and its excess may lead to inflammatory diseases and mood disorders.
Optimal range: 5 - 14.8 %
Arachidonic acid is an inflammatory omega-6 fatty acid. Our bodies produce this nutrient, and its excess may lead to inflammatory diseases and mood disorders.
Optimal range: 5.2 - 12.9 %
Arachidonic acid (AA) is a key omega-6 fatty acid measured in the OmegaCheck test offered by Cleveland HeartLab, which assesses cardiovascular risk based on polyunsaturated fatty acid (PUFA) status.
Optimal range: 5.5 - 19.01 %
Arachidonic acid (AA) is a 20-carbon polyunsaturated n-6 fatty acid with 4 double bonds (20:4n6). Its double bonds contribute to cell membrane fluidity and predispose it to oxygenation. This can lead to several important metabolites which ensure a properly functioning immune system as well as regulate inflammation, brain activity, and other signaling cascades. AA’s metabolites are called eicosanoids which are signaling molecules.
Optimal range: 0 - 250 qg/mL
Arachidonic acid is an inflammatory omega-6 fatty acid. Our bodies produce this nutrient, and its excess may lead to inflammatory diseases and mood disorders.
Optimal range: 3.7 - 40.7 Ratio
This test measures the ratio of arachidonic acid (AA) to eicosapentaenoic acid (EPA) in plasma. This ratio of the principle omega-3 and omega-6 fatty acids is a measure of the body’s eicosanoid balance. Balancing these eicosanoids in the body is an excellent way
for managing heart disease and other chronic and inflammatory processes.
Optimal range: 0 - 6 mmol/mol creatinine
Arginine is found in all protein foods and is very abundant in seeds and nuts. It is considered a semi-essential amino acid during early development, infection/inflammation, or renal and/or intestinal impairment. It has many functions in the body including:
- ammonia disposal in the urea cycle
- immune function
- stimulation of insulin release
- muscle metabolism (creatine/creatinine precursor)
- nitric oxide (NO) formation
- glutamic acid and proline formation
- glucose/glycogen conversion
- stimulation of the release of growth hormone, vasopressin, and prolactin
- wound healing
Because arginine is a precursor for nitric oxide synthesis, it is often used therapeutically in cardiovascular disease for its vasodilatory effects.
Optimal range: 0 - 26.4 nmol/mg Creatinine
→ Arginine is a precursor of urea, nitric oxide, polyamines (putrescine, spermidine, spermine and agmatine), proline, glutamate, and creatine.
→ Levels may drop with increased need, or in renal or small intestine dysfunction.
→ Synthesis of arginine depends on citrulline levels and is less regulated by dietary arginine. Supplementation with citrulline increases plasma arginine and the production of nitric oxide (NO).
→ Arginine supplementation may lower blood pressure.
→ Metabolism of elevated arginine levels is dependent on glycine availability.
→ Plasma arginine and ornithine increased, while citrulline remained stable, following watermelon juice intake, a rich source of citrulline.